Author Topic: ________ 13 _____ 2019 ________ ______ ______ HD>> 15-11-2019  (Read 172 times)

SenaidaKre

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________ 13 _____ 2019 ________ ______ ______ HD>> 15-11-2019
« on: November 14, 2019, 07:00:00 PM »
_________ 13 _____ 2019 ________ ______ ______" ________ ______


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________ 13 _____ 2019 ________ ______ ______
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HobE4ertMor

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Re: «________ 13 _____ 2019 ________ ______ ______» HD>> 15-11-2019
« Reply #1 on: October 22, 2020, 09:08:51 AM »

amiodarone 200 mg para que sirve 
ENTER

 
This medicine contains lactose. If your doctor has told you that you have an intolerance to sugars, consult him before taking it.
If you stop taking amiodarone.
Pakistan: &#9650.
Very rare side effects:
It is well known that Amiodarone Aguettant adsorbs to polyvinyl chloride (PVC) tubing and the clinical trial dose administration schedule was designed to account for this adsorption. All of the clinical trials were conducted using PVC tubing and its use is therefore recommended. The concentrations and rates of infusion provided in DOSAGE AND ADMINISTRATION reflect doses identified in these studies. It is important that the recommended infusion regimen be followed closely.
Amiodarone Aguettant injection infusions exceeding 2 hours must be administered in glass or polyolefin bottles containing D5W. Use of evacuated glass containers for admixing Amiodarone Aguettant injection is not recommended as incompatibility with a buffer in the container may cause precipitation.
This medication is used to treat certain types of serious (possibly fatal) irregular heartbeat (such as persistent ventricular fibrillation/tachycardia). It is used to restore normal heart rhythm and maintain a regular, steady heartbeat. Amiodarone Aguettant is known as an anti-arrhythmic drug. It works by blocking certain electrical signals in the heart that can cause an irregular heartbeat.
Intravenous Amiodarone Aguettant does not need to be protected from light during administration.
Respiratory function tests are usually abnormal with a restrictive pattern on spirometry and decreased gas transfer 2,6 . Hypoxemia is almost always present 6 .
treatment longer than two months age over 60 years daily dose >400 mg antecedent lung disease or surgery prior angiographic investigations.
Amiodarone hydrochloride is a triiodinated antiarrhythmic, comprised of 37% iodine by weight, which accumulates in type II pneumocytes 5,7 . As is the case with other drug-induced pulmonary toxicity, amiodarone can cause a variety of histopathologic patterns including 6-7 :
Overall pulmonary toxicity occurs in 5-10% of treated patients 4-6 .
peripheral areas of consolidation upper lobe predominance underlying interstitial disease.
In addition, the liver (80% of cases) and sometimes the heart (20%) are high density 6 . However, high hepatic and splenic attenuation is also seen in patients exposed to amiodarone in the absence of drug toxicity.
There are two main patterns of involvement, which may co-exist.
A distinctive feature of amiodarone lung is the presence of foamy histiocytes which contain intracytoplasmic osmiophilic lamellar bodies. However, this feature is also seen in patients with amiodarone exposure and no evidence of toxicity.
A unique ID assigned by the FDA when a product is submitted for approval by the labeller.
In a pharmacokinetic study of 3 healthy individuals and 3 patients diagnosed with supraventricular tachycardia (SVT), the volume of distribution was found to be 9.26-17.17 L/kg in healthy volunteers and 6.88-21.05 L/kg in the SVT patients. 6 Prescribing information mentions that the volume of distribution of amiodarone varies greatly, with a mean distribution of approximately 60 L/kg. It accumulates throughout the body, especially in adipose tissue 4 and highly vascular organs including the lung, liver, and spleen. One major metabolite of amiodarone, desethylamiodarone (DEA), is found in even higher proportions in the same tissues as amiodarone. 18.
The clearance of amiodarone after intravenous administration in patients with ventricular fibrillation and ventricular tachycardia ranged from 220 to 440 ml/hr/kg in one clinically study. 18 Another study determined that the total body clearance of amiodarone varies from 0.10 to 0.77 L/min after one intravenous dose. 4 Renal impairment does not appear to affect the clearance of amiodarone, but hepatic impairment may reduce clearance. Patients with liver cirrhosis exhibited significantly lower Cmax and mean amiodarone concentration for DEA, but not for amiodarone. Severe left ventricular dysfunction prolongs the half-life of DEA. 18.
After intravenous administration, amiodarone acts to relax smooth muscles that line vascular walls, decreases peripheral vascular resistance (afterload), and increases the cardiac index by a small amount. Administration by this route also decreases cardiac conduction, preventing and treating arrhythmias. 2,7,18 When it is given orally, however, amiodarone does not lead to significant changes in the left ventricular ejection fraction. Similar to other anti-arrhythmic agents, controlled clinical trials do not confirm that oral amiodarone increases survival. 18.
With mild asymptomatic eruptions, the history and physical examination are often sufficient for diagnosis; with severe or persistent eruptions, further diagnostic testing may be required, as follows:
Drug eruptions can mimic a wide range of dermatoses. The morphologies are myriad and include morbilliform (see the image below), urticarial, papulosquamous, pustular, and bullous. Medications can also cause pruritus and dysesthesia without an obvious eruption. A drug-induced reaction should be considered in any patient who is taking medications and who suddenly develops a symmetric cutaneous eruption. Morbilliform eruption localized to striae has been described with clindamycin. [1]
For most drug eruptions, full recovery without any complications is expected; however, the following should be noted:
Mucous membrane erosions.

 
 
 

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